Thursday, 7 February 2019

Could blocking these immune cells aid weight loss?


Scientists have uncovered a mechanism in a group of immune cells in the gut that can sway nutrient metabolism to favor fat storage over energy use.
The finding may help explain why some people remain slim despite having eating habits that cause others to gain weight.
The mechanism works when intraepithelial T cells, which are a type of immune cell that resides in the lining of the small intestine, have an active gene for the protein integrin  beta 7.
In a study paper on their work that now appears in the journal Nature, researchers at Massachusetts General Hospital and Harvard Medical School, both in Boston, MA, describe mice that do not have these particular cells as "metabolically hyperactive."
When they put mice lacking the cells on a high-fat, high-sugar diet, the animals did not develop obesity, high blood pressure, high cholesterol, heart disease, or diabetes.
"After you eat," says senior study author Filip K. Swirski, Ph.D., an associate professor of radiology who also works in the Center for Systems Biology, "your body can convert energy into heat and burn it quickly or it can convert the food into fat and store it for later use."
"These cells, which are known for their function in the immune system, also appear to play an important role in that metabolic choice," he adds.

Metabolism, fat storage, and energy use
The biochemistry of how the body handles nutrients derived from food digestion is complex and requires precise regulation.
The authors refer to "strategically positioned metabolic sensors" that direct nutrients down particular molecular pathways.
Their study identifies a pathway that prioritizes fat storage over energy use. Such a function can preserve health by ensuring that energy reserves are available during times of food scarcity.
However, for the many people who today live in societies with an abundance of high-fat, high-sugar foods, such a function is more likely to undermine health than support it.



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